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Regulation of M1‑type and M2‑type macrophage polarization in RAW264.7 cells by Galectin‑9
JCI - Altered macrophage differentiation and immune dysfunction in tumor development
Tolerance and M2 (alternative) macrophage polarization are related processes orchestrated by p50 nuclear factor κB | PNAS
NF-κB: blending metabolism, immunity, and inflammation: Trends in Immunology
Proposed mechanism of NF-κB-mediated tumour progression via effects on... | Download Scientific Diagram
NF‐κB as a central regulator of macrophage function in tumors - Biswas - 2010 - Journal of Leukocyte Biology - Wiley Online Library
Macrophage polarization in inflammatory diseases. - Abstract - Europe PMC
Angiotensin II induces RAW264.7 macrophage polarization to the M1‑type through the connexin 43/NF‑κB pathway
Frontiers | NF-κB Signaling in Macrophages: Dynamics, Crosstalk, and Signal Integration
IJMS | Free Full-Text | Anti-Inflammatory Activity of Extracts and Pure Compounds Derived from Plants via Modulation of Signaling Pathways, Especially PI3K/AKT in Macrophages | HTML
NF-κB signaling in inflammation | Signal Transduction and Targeted Therapy
Regulating the Polarization of Macrophages: A Promising Approach to Vascular Dermatosis
Frontiers | NF-κB Signaling in Macrophages: Dynamics, Crosstalk, and Signal Integration
NF-κB mediates lipopolysaccharide-induced alternative pre-mRNA splicing of MyD88 in mouse macrophages - Journal of Biological Chemistry
Macrophages regulate the progression of osteoarthritis - Osteoarthritis and Cartilage
NF-κB signaling in inflammation | Signal Transduction and Targeted Therapy
Figure 1 from IKKβ/NF-κB and the miscreant macrophage | Semantic Scholar
NF‐κB as a central regulator of macrophage function in tumors - Biswas - 2010 - Journal of Leukocyte Biology - Wiley Online Library
Counter Regulation of Spic by NF-κB and STAT Signaling Controls Inflammation and Iron Metabolism in Macrophages - ScienceDirect
Kinsenoside attenuates osteoarthritis by repolarizing macrophages through inactivating NF-κB/MAPK signaling and protecting chondrocytes - ScienceDirect