Frontiers | CDKN2A Determines Mesothelioma Cell Fate to EZH2 Inhibition
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Tri-Methyl-Histone H3 (Lys27) (C36B11) Rabbit mAb (BSA and Azide Free) | Cell Signaling Technology
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A fluorescent derivatization method of proteins for the detection of low‐level impurities by microchip capillary gel electrophoresis - Wenz - 2010 - ELECTROPHORESIS - Wiley Online Library
Tri-Methyl-Histone H3 (Lys27) (C36B11) Rabbit mAb (BSA and Azide Free) | Cell Signaling Technology
Structure-Guided Development of Small-Molecule PRC2 Inhibitors Targeting EZH2–EED Interaction | Journal of Medicinal Chemistry
Cells | Free Full-Text | Heme, A Metabolic Sensor, Directly Regulates the Activity of the KDM4 Histone Demethylase Family and Their Interactions with Partner Proteins | HTML
Biomedicines | Free Full-Text | Specific and Non-Invasive Fluorescent Labelling of Extracellular Vesicles for Evaluation of Intracellular Processing by Intestinal Epithelial Cells | HTML
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US20120135538A1 - Agent and method for identifying lysine crotonylation in proteins - Google Patents
H3K27me3 Demethylase UTX Restrains Plasma Cell Formation | The Journal of Immunology
Pleiotropic effects of the histone deacetylase Hos2 linked to H4‐K16 deacetylation, H3‐K56 acetylation, and H2A‐S129 phosphorylation in Beauveria bassiana - Cai - 2018 - Cellular Microbiology - Wiley Online Library
Tri-Methyl-Histone H3 (Lys27) (C36B11) Rabbit mAb (BSA and Azide Free) | Cell Signaling Technology
5‐Azacytidine‐mediated hMSC behavior on electrospun scaffolds for skeletal muscle regeneration - Fasolino - 2017 - Journal of Biomedical Materials Research Part A - Wiley Online Library
PDF] The Target of the NSD Family of Histone Lysine Methyltransferases Depends on the Nature of the Substrate* | Semantic Scholar
Unveiling RCOR1 as a rheostat at transcriptionally permissive chromatin | Nature Communications
Lipid Droplets Promote Phase Separation of Ago2 to Accelerate Dicer1 Loss and Decelerate miRNA Activity in Lipid Exposed Hepatic Cells | bioRxiv
Lipid Droplets Promote Phase Separation of Ago2 to Accelerate Dicer1 Loss and Decelerate miRNA Activity in Lipid Exposed Hepatic Cells | bioRxiv